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ce 10 9 cfu  (ATCC)


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    ATCC ce 10 9 cfu
    Effects of B . cellulosilyticus LYH2 and comparator treatments on disease severity, inflammation, and goblet cell number in a DSS-induced colitis mouse model. Six-week-old male C57BL/6J mice were randomly assigned to six groups (n = 7): CON (normal drinking water), DSS (3% DSS in drinking water), Ce (3% DSS and B . cellulosilyticus LYH2 at 1 × 10 9 CFU/mL), JCM (3% DSS and B . cellulosilyticus JCM 15632 at 1 × 10 9 CFU/mL), 5-ASA (3% DSS and 5-aminosalicylic acid <t>at</t> <t>100</t> mg/kg), and LR (3% DSS and Lactobacillus reuteri ATCC BAA-2837 at 1 × 10 9 CFU/mL). Bacterial strains and 5-ASA were given by oral gavage every other day for 21 days before DSS exposure. Colitis was then induced with 3% DSS for 8 days, during which bacterial treatments were paused while 5-ASA continued. (A) Body weight ratio during the experiment. (B) Final body weight. (C) DAI scores during DSS treatment. (D) Representative images of colons from each group. (E) Colon length. (F) Serum monocyte chemoattractant protein-1 (MCP-1) levels. (G) Serum interleukin-1β (IL-1β) levels. (H) Serum tumour necrosis factor-α (TNF-α) levels. (I) Serum interleukin-10 (IL-10) levels. (J) Representative H&E-stained colon sections. Scale bars = 200 μm, 10× objective. (K) Histological scores of colonic tissues. (L) Representative AB-PAS-stained colon sections. Scale bars = 200 μm, 10× objective. (M) Goblet cell number per crypt. (N) Relative mRNA expression of inflammatory and chemokine-related genes ( Tnf , Il1b , Mcp1 , and Cxcl10 ) in colon. Data are mean ± SEM (n = 7). Statistical significance was evaluated using either one-way ANOVA followed by Dunnett's post-hoc test (when normality and homogeneity of variance were met) or the Kruskal–Wallis test followed by Dunn's post-hoc test with Bonferroni correction (when these assumptions were violated), for comparisons of each group versus the DSS group. ∗ P < 0.05, ∗∗ P < 0.01, ∗∗∗ P < 0.001.
    Ce 10 9 Cfu, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 81 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/ce 10 9 cfu/product/ATCC
    Average 94 stars, based on 81 article reviews
    ce 10 9 cfu - by Bioz Stars, 2026-06
    94/100 stars

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    1) Product Images from "A next-generation probiotic strain for gut health: Bacteroides cellulosilyticus LYH2 variant with anti-inflammatory and metabolic advantages"

    Article Title: A next-generation probiotic strain for gut health: Bacteroides cellulosilyticus LYH2 variant with anti-inflammatory and metabolic advantages

    Journal: eBioMedicine

    doi: 10.1016/j.ebiom.2026.106232

    Effects of B . cellulosilyticus LYH2 and comparator treatments on disease severity, inflammation, and goblet cell number in a DSS-induced colitis mouse model. Six-week-old male C57BL/6J mice were randomly assigned to six groups (n = 7): CON (normal drinking water), DSS (3% DSS in drinking water), Ce (3% DSS and B . cellulosilyticus LYH2 at 1 × 10 9 CFU/mL), JCM (3% DSS and B . cellulosilyticus JCM 15632 at 1 × 10 9 CFU/mL), 5-ASA (3% DSS and 5-aminosalicylic acid at 100 mg/kg), and LR (3% DSS and Lactobacillus reuteri ATCC BAA-2837 at 1 × 10 9 CFU/mL). Bacterial strains and 5-ASA were given by oral gavage every other day for 21 days before DSS exposure. Colitis was then induced with 3% DSS for 8 days, during which bacterial treatments were paused while 5-ASA continued. (A) Body weight ratio during the experiment. (B) Final body weight. (C) DAI scores during DSS treatment. (D) Representative images of colons from each group. (E) Colon length. (F) Serum monocyte chemoattractant protein-1 (MCP-1) levels. (G) Serum interleukin-1β (IL-1β) levels. (H) Serum tumour necrosis factor-α (TNF-α) levels. (I) Serum interleukin-10 (IL-10) levels. (J) Representative H&E-stained colon sections. Scale bars = 200 μm, 10× objective. (K) Histological scores of colonic tissues. (L) Representative AB-PAS-stained colon sections. Scale bars = 200 μm, 10× objective. (M) Goblet cell number per crypt. (N) Relative mRNA expression of inflammatory and chemokine-related genes ( Tnf , Il1b , Mcp1 , and Cxcl10 ) in colon. Data are mean ± SEM (n = 7). Statistical significance was evaluated using either one-way ANOVA followed by Dunnett's post-hoc test (when normality and homogeneity of variance were met) or the Kruskal–Wallis test followed by Dunn's post-hoc test with Bonferroni correction (when these assumptions were violated), for comparisons of each group versus the DSS group. ∗ P < 0.05, ∗∗ P < 0.01, ∗∗∗ P < 0.001.
    Figure Legend Snippet: Effects of B . cellulosilyticus LYH2 and comparator treatments on disease severity, inflammation, and goblet cell number in a DSS-induced colitis mouse model. Six-week-old male C57BL/6J mice were randomly assigned to six groups (n = 7): CON (normal drinking water), DSS (3% DSS in drinking water), Ce (3% DSS and B . cellulosilyticus LYH2 at 1 × 10 9 CFU/mL), JCM (3% DSS and B . cellulosilyticus JCM 15632 at 1 × 10 9 CFU/mL), 5-ASA (3% DSS and 5-aminosalicylic acid at 100 mg/kg), and LR (3% DSS and Lactobacillus reuteri ATCC BAA-2837 at 1 × 10 9 CFU/mL). Bacterial strains and 5-ASA were given by oral gavage every other day for 21 days before DSS exposure. Colitis was then induced with 3% DSS for 8 days, during which bacterial treatments were paused while 5-ASA continued. (A) Body weight ratio during the experiment. (B) Final body weight. (C) DAI scores during DSS treatment. (D) Representative images of colons from each group. (E) Colon length. (F) Serum monocyte chemoattractant protein-1 (MCP-1) levels. (G) Serum interleukin-1β (IL-1β) levels. (H) Serum tumour necrosis factor-α (TNF-α) levels. (I) Serum interleukin-10 (IL-10) levels. (J) Representative H&E-stained colon sections. Scale bars = 200 μm, 10× objective. (K) Histological scores of colonic tissues. (L) Representative AB-PAS-stained colon sections. Scale bars = 200 μm, 10× objective. (M) Goblet cell number per crypt. (N) Relative mRNA expression of inflammatory and chemokine-related genes ( Tnf , Il1b , Mcp1 , and Cxcl10 ) in colon. Data are mean ± SEM (n = 7). Statistical significance was evaluated using either one-way ANOVA followed by Dunnett's post-hoc test (when normality and homogeneity of variance were met) or the Kruskal–Wallis test followed by Dunn's post-hoc test with Bonferroni correction (when these assumptions were violated), for comparisons of each group versus the DSS group. ∗ P < 0.05, ∗∗ P < 0.01, ∗∗∗ P < 0.001.

    Techniques Used: Staining, Expressing



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    ce 10 9 cfu  (ATCC)
    94
    ATCC ce 10 9 cfu
    Effects of B . cellulosilyticus LYH2 and comparator treatments on disease severity, inflammation, and goblet cell number in a DSS-induced colitis mouse model. Six-week-old male C57BL/6J mice were randomly assigned to six groups (n = 7): CON (normal drinking water), DSS (3% DSS in drinking water), Ce (3% DSS and B . cellulosilyticus LYH2 at 1 × 10 9 CFU/mL), JCM (3% DSS and B . cellulosilyticus JCM 15632 at 1 × 10 9 CFU/mL), 5-ASA (3% DSS and 5-aminosalicylic acid <t>at</t> <t>100</t> mg/kg), and LR (3% DSS and Lactobacillus reuteri ATCC BAA-2837 at 1 × 10 9 CFU/mL). Bacterial strains and 5-ASA were given by oral gavage every other day for 21 days before DSS exposure. Colitis was then induced with 3% DSS for 8 days, during which bacterial treatments were paused while 5-ASA continued. (A) Body weight ratio during the experiment. (B) Final body weight. (C) DAI scores during DSS treatment. (D) Representative images of colons from each group. (E) Colon length. (F) Serum monocyte chemoattractant protein-1 (MCP-1) levels. (G) Serum interleukin-1β (IL-1β) levels. (H) Serum tumour necrosis factor-α (TNF-α) levels. (I) Serum interleukin-10 (IL-10) levels. (J) Representative H&E-stained colon sections. Scale bars = 200 μm, 10× objective. (K) Histological scores of colonic tissues. (L) Representative AB-PAS-stained colon sections. Scale bars = 200 μm, 10× objective. (M) Goblet cell number per crypt. (N) Relative mRNA expression of inflammatory and chemokine-related genes ( Tnf , Il1b , Mcp1 , and Cxcl10 ) in colon. Data are mean ± SEM (n = 7). Statistical significance was evaluated using either one-way ANOVA followed by Dunnett's post-hoc test (when normality and homogeneity of variance were met) or the Kruskal–Wallis test followed by Dunn's post-hoc test with Bonferroni correction (when these assumptions were violated), for comparisons of each group versus the DSS group. ∗ P < 0.05, ∗∗ P < 0.01, ∗∗∗ P < 0.001.
    Ce 10 9 Cfu, supplied by ATCC, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/ce 10 9 cfu/product/ATCC
    Average 94 stars, based on 1 article reviews
    ce 10 9 cfu - by Bioz Stars, 2026-06
    94/100 stars
    		  Buy from Supplier

    Image Search Results


    Effects of B . cellulosilyticus LYH2 and comparator treatments on disease severity, inflammation, and goblet cell number in a DSS-induced colitis mouse model. Six-week-old male C57BL/6J mice were randomly assigned to six groups (n = 7): CON (normal drinking water), DSS (3% DSS in drinking water), Ce (3% DSS and B . cellulosilyticus LYH2 at 1 × 10 9 CFU/mL), JCM (3% DSS and B . cellulosilyticus JCM 15632 at 1 × 10 9 CFU/mL), 5-ASA (3% DSS and 5-aminosalicylic acid at 100 mg/kg), and LR (3% DSS and Lactobacillus reuteri ATCC BAA-2837 at 1 × 10 9 CFU/mL). Bacterial strains and 5-ASA were given by oral gavage every other day for 21 days before DSS exposure. Colitis was then induced with 3% DSS for 8 days, during which bacterial treatments were paused while 5-ASA continued. (A) Body weight ratio during the experiment. (B) Final body weight. (C) DAI scores during DSS treatment. (D) Representative images of colons from each group. (E) Colon length. (F) Serum monocyte chemoattractant protein-1 (MCP-1) levels. (G) Serum interleukin-1β (IL-1β) levels. (H) Serum tumour necrosis factor-α (TNF-α) levels. (I) Serum interleukin-10 (IL-10) levels. (J) Representative H&E-stained colon sections. Scale bars = 200 μm, 10× objective. (K) Histological scores of colonic tissues. (L) Representative AB-PAS-stained colon sections. Scale bars = 200 μm, 10× objective. (M) Goblet cell number per crypt. (N) Relative mRNA expression of inflammatory and chemokine-related genes ( Tnf , Il1b , Mcp1 , and Cxcl10 ) in colon. Data are mean ± SEM (n = 7). Statistical significance was evaluated using either one-way ANOVA followed by Dunnett's post-hoc test (when normality and homogeneity of variance were met) or the Kruskal–Wallis test followed by Dunn's post-hoc test with Bonferroni correction (when these assumptions were violated), for comparisons of each group versus the DSS group. ∗ P < 0.05, ∗∗ P < 0.01, ∗∗∗ P < 0.001.

    Journal: eBioMedicine

    Article Title: A next-generation probiotic strain for gut health: Bacteroides cellulosilyticus LYH2 variant with anti-inflammatory and metabolic advantages

    doi: 10.1016/j.ebiom.2026.106232

    Figure Lengend Snippet: Effects of B . cellulosilyticus LYH2 and comparator treatments on disease severity, inflammation, and goblet cell number in a DSS-induced colitis mouse model. Six-week-old male C57BL/6J mice were randomly assigned to six groups (n = 7): CON (normal drinking water), DSS (3% DSS in drinking water), Ce (3% DSS and B . cellulosilyticus LYH2 at 1 × 10 9 CFU/mL), JCM (3% DSS and B . cellulosilyticus JCM 15632 at 1 × 10 9 CFU/mL), 5-ASA (3% DSS and 5-aminosalicylic acid at 100 mg/kg), and LR (3% DSS and Lactobacillus reuteri ATCC BAA-2837 at 1 × 10 9 CFU/mL). Bacterial strains and 5-ASA were given by oral gavage every other day for 21 days before DSS exposure. Colitis was then induced with 3% DSS for 8 days, during which bacterial treatments were paused while 5-ASA continued. (A) Body weight ratio during the experiment. (B) Final body weight. (C) DAI scores during DSS treatment. (D) Representative images of colons from each group. (E) Colon length. (F) Serum monocyte chemoattractant protein-1 (MCP-1) levels. (G) Serum interleukin-1β (IL-1β) levels. (H) Serum tumour necrosis factor-α (TNF-α) levels. (I) Serum interleukin-10 (IL-10) levels. (J) Representative H&E-stained colon sections. Scale bars = 200 μm, 10× objective. (K) Histological scores of colonic tissues. (L) Representative AB-PAS-stained colon sections. Scale bars = 200 μm, 10× objective. (M) Goblet cell number per crypt. (N) Relative mRNA expression of inflammatory and chemokine-related genes ( Tnf , Il1b , Mcp1 , and Cxcl10 ) in colon. Data are mean ± SEM (n = 7). Statistical significance was evaluated using either one-way ANOVA followed by Dunnett's post-hoc test (when normality and homogeneity of variance were met) or the Kruskal–Wallis test followed by Dunn's post-hoc test with Bonferroni correction (when these assumptions were violated), for comparisons of each group versus the DSS group. ∗ P < 0.05, ∗∗ P < 0.01, ∗∗∗ P < 0.001.

    Article Snippet: Effects of B . cellulosilyticus LYH2 and comparator treatments on disease severity, inflammation, and goblet cell number in a DSS-induced colitis mouse model. Six-week-old male C57BL/6J mice were randomly assigned to six groups (n = 7): CON (normal drinking water), DSS (3% DSS in drinking water), Ce (3% DSS and B . cellulosilyticus LYH2 at 1 × 10 9 CFU/mL), JCM (3% DSS and B . cellulosilyticus JCM 15632 at 1 × 10 9 CFU/mL), 5-ASA (3% DSS and 5-aminosalicylic acid at 100 mg/kg), and LR (3% DSS and Lactobacillus reuteri ATCC BAA-2837 at 1 × 10 9 CFU/mL).

    Techniques: Staining, Expressing